Quit Dipping/How To Quit/Resource Library/E-Cigarettes: What's the Harm in Vapor?From: To: CC: Subject: Body: E-Cigarettes: What's the Harm in Vapor?Electronic cigarettes have become a hot item.Since they first appeared in China in 2004 and began to be imported, the numbers of brands and users have jumped.Many smokers have been drawn to claims that e-cigarettes are a safe substitute for standard cigarettes because they deliver nicotine without the harmful chemicals and toxins in tobacco smoke.Some e-cigarette supporters say these products can help smokers to quit or cut down the amount of tobacco they use.What’s more, e-cigarettes supposedly do not expose others to the dangers of second-hand smoke.Yet many health experts have sounded alarms about e-cigarettes, calling for more study and control.Much of the debate has been about nicotine, the key—and addictive—ingredient in most e-cigarettes.Issues have included uneven delivery of inhaled nicotine, quality control problems such as poor labeling and leaks that can expose e-cigarette users to toxic liquid nicotine, maintaining a person’s addiction to nicotine, and uncertainty about effects on health over time.

Until recently, the vapor that puffs from e-cigarettes in place of tobacco smoke has drawn little attention.e cigarette buttonIn fact, many fans of e-cigs say that, because they create a mist produced by propylene glycol or vegetable glycerin rather than smoke, they should not be covered by no-smoking rules in public places.e cigarette bondiBut now doctors have raised concerns about e-cigarette vapor separate from those about nicotine.e cigarette barnA 42-year-old woman who had used e-cigarettes for seven months experienced shortness of breath, cough, and fever over the same period.e-cigarette create healthy life refills

The diagnosis was lipoid pneumonia—a chronic inflammation caused by fatty substances (lipids) in the lungs—due to e-cigarette use.e cigarette cognacThe specific cause was repeated exposure to glycerin-based oils found in e-cigarette nicotine vapor.e cigarette chorleyThe patient’s symptoms improved after she stopped using e-cigarettes.1 A recent study also found that using an e-cigarette for five minutes began to restrict lung function.It was not clear which e-cigarette ingredient or combination of substances caused these reactions.However, other studies have shown that propylene glycol, the source of the e-cigarettes’ visible vapor, can bring on respiratory irritation and increase the chance of developing asthma.2 These new findings are quite limited, but they are reminders that the e-cigarette story is still unfolding with many questions yet to be answered.

One such question in addition to the effects of e-cigarettes on users is whether secondhand vapors may place other people at risk.Our advice: proceed with caution and stay on top of the latest findings on e-cigs.To find out more, check out the Electronic Cigarettes section in our Resource Library section. is here to support you.References 1 McCauley, L., Markin, C., & Hosmer, D.An Unexpected Consequence of Electronic Cigarette Use.2 Vardavas, C.I., Anagnostopoulos, N., Kougias, M., Evangelopoulou, V., Connolly, G.N., & Behrakis, P.K.Acute pulmonary effects of using an e-cigarette: Impact on respiratory flow resistance, impedance and exhaled nitric oxide.Access the info you need to help you live a tobacco-free life.Sign up for SmokefreeMIL and get quit support texts.Epub 2016 Jan 6.Dicpinigaitis PV1, Lee Chang A2, Dicpinigaitis AJ2, Negassa A3.Author information1Department of Medicine, Albert Einstein College of Medicine, Bronx, NY; Department of Medicine, Montefiore Medical Center, Bronx, NY.

.2Department of Medicine, Albert Einstein College of Medicine, Bronx, NY; Department of Medicine, Montefiore Medical Center, Bronx, NY.3Department of Epidemiology and Population Health, Albert Einstein College of Medicine, Bronx, NY.AbstractBACKGROUND: E-cigarettes (e-cigs) have attained widespread popularity, yet knowledge of their physiologic effects remains minimal.The aim of this study was to evaluate the effect of a single exposure to e-cig vapor on cough reflex sensitivity.METHODS: Thirty healthy nonsmokers underwent cough reflex sensitivity measurement using capsaicin cough challenge at baseline, 15 min, and 24 h after e-cig exposure (30 puffs 30 s apart).The end point of cough challenge is the concentration of capsaicin inducing five or more coughs (C5).The number of coughs induced by each e-cig inhalation was counted.A subgroup of subjects (n = 8) subsequently underwent an identical protocol with a non-nicotine-containing e-cig.RESULTS: Cough reflex sensitivity was significantly inhibited (C5 increased) 15 min after e-cig use (?0.29; 95% CI, ?0.43 to ?0.15; P < .0001); 24 h later, C5 returned to baseline (0.24; 95% CI, 0.10-0.38; P = .0002 vs post-15-min value).

A subgroup of eight subjects demonstrating the largest degree of cough reflex inhibition had no suppression after exposure to a non-nicotine-containing e-cig (P = .0078 for comparison of ?C5 after nicotine vs non-nicotine device).Furthermore, more coughing was induced by the nicotine-containing vs non-nicotine-containing device (P = .0156).CONCLUSIONS: A single session of e-cig use, approximating nicotine exposure of one tobacco cigarette, induces significant inhibition of cough reflex sensitivity.Exploratory analysis of a subgroup of subjects suggests that nicotine is responsible for this observation.Our data, consistent with previous studies of nicotine effect, suggest a dual action of nicotine: an immediate, peripheral protussive effect and a delayed central antitussive effect.TRIAL REGISTRY: ClinicalTrials.gov; No.: NCT02203162; URL: www.clinicaltrials.gov.Copyright © 2016 American College of Chest Physicians.Published by Elsevier Inc.All rights reserved.KEYWORDS: capsaicin; cough; electronic cigarette; nicotinePMID: 26291648 DOI: 10.1378/chest.15-0817 [Indexed for MEDLINE] Publication typeControlled Clinical TrialMeSH termsAdultCapsaicinCough/etiology*Cough/physiopathology*Electronic Cigarettes/adverse effects*FemaleGanglionic Stimulants/pharmacology*HumansMaleNicotine/pharmacology*Reflex/drug effects*Reflex/physiologySensory System AgentsSubstancesGanglionic StimulantsSensory System AgentsNicotineCapsaicinSecondary source IDClinicalTrials.gov/NCT02203162Full Text SourcesElsevier ScienceSilverchair Information SystemsOvid Technologies, Inc.MedicalClinicalTrials.govE-Cigarettes - MedlinePlus Health InformationCough - MedlinePlus Health InformationMiscellaneousCAPSAICIN - Hazardous Substances Data BankNICOTINE - Hazardous Substances Data BankNCI CPTAC Assay Portal